Atypical Absence Seizure

Overview of Atypical Absence Seizure

Most recent studies have shown that Atypical Absence Seizure shares some biological mechanisms with absence-epilepsy, atonic-seizures, complex-partial-seizures, convulsive-seizures, encephalopathies, epilepsies-myoclonic, epilepsies-partial, epilepsy, epilepsy-generalized, generalized-seizures, infantile-spasms, intractable-epilepsy, lennox-gastaut-syndrome, partial-seizure, spasm, status-epilepticus, tonic-clonic-epilepsy, tonic-convulsion.

Among the many pathways, these few ones have gauged particular interests from scientists studying Atypical Absence Seizure, and have been seen in publications frequently: Aging, Brain Development, Cell Proliferation, Cognition, Glucose Transport, Neurogenesis, Pathogenesis, Proestrus, Reflex, Synaptic Transmission, Transport

Quite a number of genes have been found to play important roles in Atypical Absence Seizure, such as BCL9, ERG, EXOSC7, EXT1, FGD1, FOS, FSCN1, HTN3, IL2RA, MCAT, MPP1, NUPR1, P4HB, POLG2, PSMD12, S100A8, SCN1A, SPINT2, TNFRSF1A, TPPP3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Atypical Absence Seizure Related Genes

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Pathways Related to Atypical Absence Seizure

This information is being compiled and will come in a future update

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